The human eye has three kinds of cone cells that respond differently depending on the wavelength of light: long (L, toward red), medium (M, toward green), and short (S, toward blue). When one of these cone types is missing (color blindness, or dichromacy) or its sensitivity is shifted (color weakness, or anomalous trichromacy), certain colors become hard to tell apart.
The color strip for each type shows the same ten colors simulated through that type's eyes (Machado 2009 model). It may differ from reality depending on your monitor.
10 reference colors as seen with normal color vision
Red-green (protan and deutan) — a problem with the L or M cones. Inherited on the X chromosome, it is far more common in men and accounts for the vast majority of color vision deficiencies. Inheritance guide
Blue-yellow (tritan) — a problem with the S cones. Inherited in an autosomal dominant pattern on chromosome 7, it affects men and women about equally and is very rare. It can also be acquired through eye disease or aging.
Monochromacy (achromatopsia and blue cone monochromacy) — almost no cones work, or only one type does. Color discrimination is nearly absent, and reduced visual acuity, glare sensitivity, and nystagmus often come with it.
Names vary by country and field. In English, “color blindness” is the everyday umbrella term — it usually refers to red-green deficiency and covers mild cases too — while clinicians and researchers prefer “color vision deficiency” (CVD), reserving “-anopia” for dichromacy and “-anomaly” for anomalous trichromacy.